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Tibor Valyi-Nagy, MD, PhD
Professor of Pathology, Director of Neuropathology

phone: (312) 996-1772

fax: (312) 996-7586


  • Education
    • MD, Medical University of Debrecen, Hungary
      PhD, Hungarian Academy of Sciences
  • Residency Training
    • Anatomic Pathology and Neuropathology, Vanderbilt University School of Medicine, Nashville, Tennessee
      Medical Microbiology, National Institute of Dermatology and Venereology, Budapest, Hungary
  • Fellowship
    • Virology and Immunology, University of Texas Medical Branch at Galveston, Texas
      Neurovirology, The Wistar Institute, Philadelphia, Pennsylvania
  • Certification
    • Anatomic Pathology and Neuropathology, American Board of Pathology
      Medical Microbiology, Hungarian Board of Medical Microbiology
  • Clinical Focus
    • Neuropathology
  • Research Focus
    • Pathogenesis of nervous system infections, herpes simplex virus pathogenesis
  • Selected Recent Publications
    • Valyi-Nagy K, Dosa S,  Kovacs SK, Bacsa S, Voros A, Shukla D, Folberg D, Valyi-Nagy T. Identification of virus resistant tumor cell subpopulations in three dimensional uveal melanoma cultures. Cancer Gene Therapy, 17:223-234, 2010.

      Dosa S, Castellanos K, Bacsa S, Gagyi E, Kovacs SK, Valyi-Nagy K, Shukla D, Dermody TS, Valyi-Nagy T. Chronic progressive deficits in neuron size, density, and number in the trigeminal ganglia of mice latently infected with herpes simplex virus. Brain Pathology, 21:583-593, 2011.

      Gagyi E, Kormos B, Castellanos KJ, Valyi-Nagy K, Korneff D,  LoPresti P, Woltjer R, Valyi-Nagy T. Decreased oligodendrocyte nuclear diameter in Alzheimer’s disease and Lewy body dementia. Brain Pathology, 22:803-810, 2012.

      Shukla ND, Tiwari V, Valyi-Nagy T. Nectin-1-specific entry of herpes simplex virus 1 is sufficient for infection of the cornea and viral spread to the trigeminal ganglia. Molecular Vision, 18:2711-2716, 2012.

      Castellanos KJ, Gagyi E, Kormos B, Valyi-Nagy K, Voros A, Shukla D, Horvath S, Slavin KV, Valyi-Nagy T. Increased axonal expression of nectin-1 in multiple sclerosis plaques. Neurological Sciences, 34:465-469, 2013.